Re: Life's Lethal Quality Control?

From: Damien Broderick (damienb@unimelb.edu.au)
Date: Thu Sep 04 2003 - 00:21:13 MDT

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    >> Not so -- K-selection vs. R-selection will apply selection pressure
    >> particularly if the "elders" of a group contribute significantly
    >> to the survival of their children, grand-children, etc. This seems
    >> likely to be true in humans, elephants and whales -- though there
    >> is still a fair amount of academic debate on this topic.

    >Yes, I see your point here. I hadn't considered that. I keep forgetting that
    >"social" aspect of our nature. Maybe because I'm reclusive. :-)
    >Still, I have to wonder what, if any, contribution "elders" would have that
    >were significant enough to increase the likelihood of grand-children
    >surviving to reproductive age in the early hunter-gatherer tribes of modern
    >humans.

    According to that splendid book THE LAST MORTAL GENERATION (which might be
    out of date by now, four years later):

    ========

            Die young, and leave a beautiful child

    Menopause - `the Change' - occurs, you'll recall, when pituitary hormones
    fail to activate repair and maintenance systems further down the track. It
    seems an absurdly early point in human life to shut down the reproductive
    system, since menopause begins thirty-odd years before many modern women
    are ready to die of old age. Surely the Darwinian dogma tells us that the
    sterile lose their place in the evolutionary queue. However healthy they
    happen to be, their success as phenotypes (or bodies) cannot win their
    genes a place in the next generation. They might even find fertile mates,
    but they have already lost the contest. There are well-known exceptions to
    this harsh economics, mostly among insects where sterile workers are
    selected because they share more than the usual proportion of genes with
    their fertile sisters, and aid the hive's prosperity. Humans don't have
    that advantage - except on a cultural level, where single celibates,
    dedicated homosexuals and other `non-breeders' enrich the survival chances
    of their child-bearing fellows. Is that the clue we need to explain
    menopause's peculiarities?
            Recall that the endocrine cascade of menopause starts when the last of the
    egg follicles in a woman's ovaries run out. This brute fact can be
    interpreted in a several ways, some of them profoundly misleading. Without
    eggs, it is sometimes said, a woman is no longer at the sharp end of the
    natural selection process, so she can be discarded - left to die,
    literally. This, as we'll see, is exactly what happens to other mammals
    such as baboons and lions. A more accurate way of putting it is that, in
    practice, bodies built from genes which managed to keep them healthy and
    nimble until they were a thousand years old would have no reproductive
    value over bodies that drop dead of heart attacks at 50 - if they cease
    passing those genes on to descendants at 45. Evolution is a sieve for
    groups of genes, not for bodies as such.
            One trouble with that stark account, which seems sound as far as it goes,
    is that it neglects the contribution of post- or non-reproductive family
    members who share the same genes. Think of two imaginary human tribes, each
    with several inter-marrying bands, who differ genetically in this respect.
    In one, mothers simply die at about 50 or 55, seven years after giving
    birth to their last child, conceived from one of their last eggs. The
    moment the youngest child in the family is reasonably self-sufficient, the
    mother (due to her characteristic blend of uncaring genes) gets cancer or
    heart disease and dies. In the other tribe, mothers remain reasonably
    healthy after an uncomfortable transition through menopause, and continue
    as productive members of their community, providing food and, as custodians
    of wisdom, hoarding and dispensing useful information that can make all the
    difference between life and death to many of her grandchildren and nieces
    and nephews for a couple more decades.
            It seems intuitively obvious that the second evolutionary strategy will
    prove more successful. Indeed, it is embodied in a recent anthropological
    doctrine known as `the Grandmother hypothesis', advanced in 1997 by Kristen
    Hawkes and James O'Connell, of the University of Utah, and Nicholas Blurton
    Jones, of UCLA, based on research among the three hundred Hadza
    hunter-gatherers in hill country near Lake Eyasi in northern Tanzania. The
    healthy Hadza grandmothers foraged for food shared by their grandchildren,
    which permitted their daughters to bear and suckle new babies rather than
    expend energy and effort looking after their older children. It is true
    that acting as wise elder repositories of knowledge (memes, in short) would
    benefit the whole tribe indiscriminately. Hence, this benefit might not
    have a direct and preferential effect on genetic reinforcement in the
    blood-lineage along, and might not be selected for. Still, providing care
    and food for their daughters' children certainly would benefit their own
    gene line.
            This is an endearing notion, and useful in offsetting the bombastic stress
    some male anthropologists have placed on the supposedly supreme virtue of
    hunting as a source of food (not that this has been taken terribly
    seriously for decades anyway). The problem with the grandmother theory,
    alas, is that it just doesn't seem to make mathematical sense. And in
    genetics, that is a very important hurdle. There's nothing abstract and
    arbitrary about the mathematical models used to test population genetics.
    The equations are clear and unforgiving, if used appropriately.
            In September, 1997, Natalie Angier reported in The New York Times that
    `Using mathematical models, Alan Rogers of the University of Utah estimated
    that a postmenopausal woman would have to double the number of children her
    children bore, and eliminate infant mortality among those grandchildren, to
    make menopause look like a sound strategy for propagating one's genes.'
    That's an improbably improvement in life chances for any woman's
    descendants, even a hardy Hadza's. I suspect, therefore, that in this raw
    form, at least, the grandmother hypothesis has failed the test.
            On the other hand, George C. Williams' model of inclusive fitness might
    yet save the day. I find it impossible to believe that the presence of at
    least some wise elders would make little or no difference to the survival
    prospects of a typical foraging tribe. Today, sadly, that's not really
    true. It is surely nice for small urban children to have the loving
    attention of their grannies, and convenient for hard-working parents to be
    able to turn over some of the task of child-minding. But none of this
    worthy contribution makes any difference, I think, to the number of
    children who survive to maturity and pass along the family's genes.
    Everything has got much more complex and confused with the coming of
    contraception, abortion choice, smaller families. If anything, it's those
    with the least resources and most disrupted family arrangements who are
    likely to have a larger brood.
            Among other species, the evidence is ambiguous. A 1998 article in Nature
    by Craig Packer, a biologist at the University of Minnesota, St. Paul, Marc
    Tatar of Brown University in Providence, Rhode Island, and Antony Collins
    of the Gombe Stream Research Centre in Kigoma, Tanzania, noted that:

            reproductive cessation has also been documented in non-human primates,
    rodents, whales, dogs, rabbits, elephants and domestic livestock. The human
    menopause has been considered an evolutionary adaptation, assuming that
    elderly women avoid the increasing complications of continued childbirth to
    better nurture their current children and grandchildren. But an abrupt
    reproductive decline might be only a non-adaptive by-product of
    life-history patterns... a systematic test of these alternatives us[ed]
    field data from two species in which grandmothers frequently engage in
    kin-directed behaviour. Both species show abrupt age-specific changes in
    reproductive performance that are characteristic of menopause. But elderly
    females do not suffer increased mortality costs of reproduction, nor do
    post-reproductive females enhance the fitness of grandchildren or older
    children. Instead, reproductive cessation appears to result from senescence.

    The animals studied were baboons and lions. The distinction in life span is
    revealing. Female baboons who make it to old age become menopausal at 21,
    and survive an extra five years - a couple of years longer than needed for
    their last infant to outgrow dependency. Female lions live only 18 years,
    with menopause at 14, and their cubs need about a year of maternal attention.
            Kristen Hawkes argues that this might not be as water-tight an argument
    against the `Grandmother Hypothesis' as it seems, in evolutionary terms.
    Four-fifths of hunter-gatherer women, these days at least, survive beyond
    menopause, often into their seventies, so their contribution is marked.
    Lions, though, average only an extra three and a half years, and baboons
    only five.

            Tried and true

    The basic argument here is that an organism that has stopped bearing young
    - or, in the elaborated version, can no longer contribute to the thriving
    of grandchildren - loses its relevance in the genetic struggle for
    survival. That is, we die after we have fallen too far behind in the
    struggle for genetic endurance. Accumulating errors exceed the ability of
    repair systems to proofread fix them, even (to a lesser extent) the sex
    cells. But this analysis, as we have seen, assumes part of what needs to be
    demonstrated - that older organisms automatically cease breeding reliably.
    While that is contingently true of complex creatures, as we have just seen,
    what we need to ask is: why it should be so?
            In other words, it is logically possible that an endlessly healthy,
    self-repairing and fertile couple could run a production-line of their
    joint genes until the end of time, with an inclusive fitness function (in
    the jargon of the population geneticists) for their very own selfish genes
    which is superior to that of their rivals' gene pool. Both Darwin and
    Mendel would rub their hands with glee without a single censorious word of
    reproof. The only reason our earlier imaginary tribes came to the same
    grisly end was that the thousand-year-old women in the long-lived tribe
    were assumed, rather arbitrarily, to become infertile at about 45.
            Why, after all, shouldn't the machinery of the body's maintenance systems
    keep working at peak efficiency forever? The accepted answer is that there
    are better strategic trade-offs for available energy and coding, trade-offs
    that allow the old to wear out and die. One reason this happens is that the
    world is a dangerous place. It's a lethal place, most of the time, in the
    wild - and the wild is where most humans have spent their time, until very
    recently indeed.
            Another reason is that its deadliness keeps changing lanes, as viruses,
    bacteria and parasites frantically mutate. Sex happens to be a quite
    effective method for generating novel immune defence codes, blending two
    working systems into a fresh version, perhaps with some novel benefits.
    That makes sex a kind of genetic-immunological luck dip, since many
    combinations are just as likely to make the creature they specify worse off
    - but evolution is a blind process, not a caring designer, and if one
    strategy is more successful than another, it is preserved, at whatever the
    cost to individuals.

    ========

    Damien Broderick



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