>>HIV seems equally distributed between the sexes, yet males make up
>>almost 9 out of 10 AIDS cases. Paradoxical to the theory that HIV
>>causes AIDS.
Steve Edwards, former virologist, said:
>You seem to think that the US is the only country with an AIDs problem.
I never said that. I have pointed out that males make up roughly 9 out
of 10 of the American and European AIDS cases. Where did I say that
the US is the only country with an AIDS problem?
>AIDs is far more prevalent in Africa, where IV drugs are rarely used, and,
>if you believe the Africans, homosexual behavior is less common. In Africa,
>AIDS is distributed equally between the sexes.
If you look at global data, AIDS looks like different epidemics.
In the US and Europe AIDS is not evenly spread among the population,
almost 90% of the patients being male. In Africa it is spread evenly among the
population and the symptoms are completely different.
If AIDS is caused by HIV, the ratio of infected to diseased carriers should
be similar in different countries. In the US roughly 10% of 1 million HIV
positives have developed AIDS since 1985. However in Uganda only 0.8%
of 1 million, and in Zaire only 0.15% of 3 million HIV positives have developed
AIDS. This indicates that the AIDS risk of infected Americans is 10-65 fold
higher than Africans. If AIDS equaled opportunistic infections resulting
from immunosupression, MORE, instead of less, AIDS per HIV carrier
should occur in Africa vs the US.
Also, all infected subjects should have the same risk of developing those
AIDS diseases that are not associated with group or region specific parasites.
53% of American AIDS patients have pneumocystis pneumonia and
13% candidiasis. 90% of the AIDS patients in Africa have slim disease,
fever, diarrhea, and tuberculosis but not pneumonia or candidiasis,
although the microbes that cause Pneumonia and candidiasis are
ubiquitous in humans, including Africans.
Rather than being reassured that the statistical anomalies
(for an infectious disease) of American AIDS can be averaged out with
data from other countries, I wonder if the categorically different
epidemiology is a function of their different causes. After all,
a 50-50% distribution by sex vs a 90-10% distribution is as
different as night and day, epidemiologically.
In Africa, Duesberg identifies parasitic diseases, poor sanitation and
malnutrition as the main causes of AIDS. In the US and Europe, he holds
that toxic drugs or conventional clinical deficiencies and their treatments
cause the indicator diseases of AIDS.
This hypothesis resolves many of the paradoxes of the virus-AIDS dogma.
1)The difference in the epidemiology of African vs American AIDS.
2)The prevalence of AIDS in American males despite evidence that both
males and females are equally infected by HIV.
3)The vastly different AIDS diseases are caused by different pathogens,
pathogenic conditions and their treatments.
4)The fact that hemophiliacs have doubled their median age in the last
10-15 years, while 75% of them have been infected with HIV for almost 10 yrs.
4)The long and unpredictable latent periods between infection by HIV
and specific AIDS diseases are the product of functually unrelated events:
the pathogenic events necessary to reach an individual's threshold for
AIDS diseases, and infection by the marker HIV.
>Some persistent viruses can be dormant for years before
>causing disease. Herpes can cause disease only periodically even though
>the patient is chronically infected.
Herpes simplex virus can cause lesions even long after the first antibodies
against the virus have been produced. But this can only happen if the virus
is reactivated because the origianl antibodies and anti-viral T-cells have
dropped below a safe threshold level. After reactivation the virus multiplies
into large numbers just as in the original infection.
But herpes produces the same lesions upon first infecting the body as it does
upon reactivation, and antibodies neutralize it both early and late. Herpes can
only recur because it hides in certain cells, waiting until some future
opportunity when the host's immune function is temporarily reduced. Once
the immune system regains strength, the virus is again suppressed and the
sores disappear.
When herpes is causing lesions it is active and abundent and found in the
diseased tissues. HIV, on the other hand, only infects a tiny % of the T-cells
while the immune system is being damaged. Herpes causes the
same symptoms upon first infecting the body and upon reactivation.
HIV is alleged to kill its host only years after being neutralized, and even
without reactivating. Thus herpes is an inappropriate model for HIV/AIDS.
Regards,
Pat Fallon
PFallon@bigfoot.com